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<SEC-DOCUMENT>0001075880-08-000035.txt : 20080506
<SEC-HEADER>0001075880-08-000035.hdr.sgml : 20080506
<ACCEPTANCE-DATETIME>20080505192130
ACCESSION NUMBER:		0001075880-08-000035
CONFORMED SUBMISSION TYPE:	6-K
PUBLIC DOCUMENT COUNT:		2
CONFORMED PERIOD OF REPORT:	20080415
FILED AS OF DATE:		20080506
DATE AS OF CHANGE:		20080505

FILER:

	COMPANY DATA:	
		COMPANY CONFORMED NAME:			NOVOGEN LTD
		CENTRAL INDEX KEY:			0001075880
		STANDARD INDUSTRIAL CLASSIFICATION:	PHARMACEUTICAL PREPARATIONS [2834]
		IRS NUMBER:				000000000
		FISCAL YEAR END:			0630

	FILING VALUES:
		FORM TYPE:		6-K
		SEC ACT:		1934 Act
		SEC FILE NUMBER:	000-29962
		FILM NUMBER:		08804251

	BUSINESS ADDRESS:	
		STREET 1:		140 WICKS RD
		STREET 2:		NORTH RYDE
		CITY:			NEW SOUTH WALES 2113
		STATE:			C3
		ZIP:			2113
		BUSINESS PHONE:		01161298780088

	MAIL ADDRESS:	
		STREET 1:		107 NORTH RYDE
		CITY:			SYDNEY
		STATE:			C3
		ZIP:			1670
</SEC-HEADER>
<DOCUMENT>
<TYPE>6-K
<SEQUENCE>1
<FILENAME>form6k20080415.htm
<DESCRIPTION>FORM 6K 2008 04 15
<TEXT>
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cover of Form 20-F or Form 40-F.</font></div>
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by check mark if the registrant is submitting the Form 6-K in paper as permitted
by Regulation S-T Rule 101(b)(l):<font style="DISPLAY: inline; FONT-WEIGHT: bold">&#160;</font><font style="DISPLAY: inline; FONT-WEIGHT: bold;" face="Wingdings">o</font></font></div>
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Regulation S-T Rule 101 (b)( I) only permits the submission in paper of a Form
6-K if submitted solely to provide an attached annual report to security
holders.</font></div>
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by check mark if the registrant is submitting the Form 6-K in paper as permitted
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Regulation S-T Rule l01(b)(7) only permits the submission in paper of a Form 6-K
if submitted to furnish a report or other document that the registrant foreign
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in which the registrant is incorporated, domiciled or legally organized (the
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which the registrant&#8217;s securities are traded, as long as the report or other
document is not a press release, is not required to be and has not been
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filing on EDGAR.</font></div>
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    <div style="DISPLAY: block; MARGIN-LEFT: 0pt; TEXT-INDENT: 0pt; MARGIN-RIGHT: 0pt; TEXT-ALIGN: left" align="justify"><font style="DISPLAY: inline; FONT-SIZE: 14pt; FONT-FAMILY: Times New Roman"><font style="DISPLAY: inline; FONT-WEIGHT: bold">Pursuant to the requirements of the
Securities Exchange Act of 1934, the registrant has duly caused this report to
be signed on its behalf </font>by the undersigned, thereunto duly
authorized.</font></div>
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Limited</font></div>
    <div style="DISPLAY: block; MARGIN-LEFT: 0pt; TEXT-INDENT: 0pt; MARGIN-RIGHT: 0pt; TEXT-ALIGN: left" align="left"><font style="DISPLAY: inline; FONT-SIZE: 14pt; FONT-FAMILY: Times New Roman">(Registrant)</font></div>
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Erratt</font></div>
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Lea Erratt</font></div>
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Secretary</font></div>
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15 April, 2008</font></div>
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    <div style="TEXT-ALIGN: right"><img src="novogenletterhead.jpg" alt="Novogen Logo"></div>
    <div>&#160;</div>
    <div>&#160;</div>
    <div>&#160;</div>
    <div>
      <div style="DISPLAY: block; MARGIN-LEFT: 0pt; TEXT-INDENT: 0pt; MARGIN-RIGHT: 0pt" align="left"><font style="DISPLAY: inline; FONT-WEIGHT: bold; FONT-SIZE: 14pt; FONT-FAMILY: Times New Roman">ASX
&amp; MEDIA RELEASE</font></div>
      <div style="DISPLAY: block; MARGIN-LEFT: 0pt; TEXT-INDENT: 0pt; MARGIN-RIGHT: 0pt" align="left"><font style="DISPLAY: inline; FONT-WEIGHT: bold; FONT-SIZE: 14pt; FONT-FAMILY: Times New Roman">15
APRIL, 2008</font></div>
      <div style="DISPLAY: block; TEXT-INDENT: 0pt"><br></div>
      <div style="DISPLAY: block; MARGIN-LEFT: 0pt; TEXT-INDENT: 0pt; MARGIN-RIGHT: 0pt" align="left"><font style="DISPLAY: inline; FONT-WEIGHT: bold; FONT-SIZE: 14pt; FONT-FAMILY: Times New Roman">NOVOGEN&#8217;S
NV-128 IS A UNIQUE INHIBITOR OF mTOR DEPHOSPHORYLATION LEADING TO CASPASE
INDEPENDENT DEATH IN CHEMORESISTANT CANCER CELLS</font></div>
      <div style="DISPLAY: block; MARGIN-LEFT: 0pt; TEXT-INDENT: 0pt; MARGIN-RIGHT: 0pt" align="justify">&#160;</div>
      <div style="DISPLAY: block; MARGIN-LEFT: 0pt; TEXT-INDENT: 0pt; MARGIN-RIGHT: 0pt" align="left"><font style="DISPLAY: inline; FONT-SIZE: 14pt; FONT-FAMILY: Times New Roman">San Diego
&#8211; 15 April - Pre-clinical studies reviewed during an oral presentation here
today at the annual meeting of the American Association for Cancer Research,
demonstrate that the Novogen drug candidate NV-128 engages a novel mode of cell
death targeting the akt-mTOR pathway in multi-drug resistant ovarian cancer
cells.&#160;&#160;The data were presented by Dr Ayesha Alvero, MD, and Associate
Professor Gil Mor, MD, Department of Obstetrics and Gynecology, Yale University
School of Medicine.</font></div>
      <div style="DISPLAY: block; MARGIN-LEFT: 0pt; TEXT-INDENT: 0pt; MARGIN-RIGHT: 0pt" align="justify">&#160;</div>
      <div style="DISPLAY: block; MARGIN-LEFT: 0pt; TEXT-INDENT: 0pt; MARGIN-RIGHT: 3.6pt" align="left"><font style="DISPLAY: inline; FONT-SIZE: 14pt; FONT-FAMILY: Times New Roman">NV-128 is
unique in that it does not induce caspase-mediated apoptosis which can be
non-functional in chemoresistant cancer cells due to accumulated mutations in
tumour suppressor/promoter genes and over-expression of anti-apoptotic
proteins.&#160;&#160;Rather, NV-128 uncouples the akt-mTOR&#173;P70S6K signal
transduction cascade which has a key role in driving protein translation and
uncontrolled cancer cell proliferation.&#160;&#160;Further, NV-128 induces
mitochondrial depolarization via a novel pathway involving the autophagy protein
Beclin-1 and Bcl-2, thereby resulting in endonuclease G translocation to the
nucleus and cell death.</font></div>
      <div style="DISPLAY: block; MARGIN-LEFT: 0pt; TEXT-INDENT: 0pt; MARGIN-RIGHT: 0pt" align="justify">&#160;</div>
      <div style="DISPLAY: block; MARGIN-LEFT: 0pt; TEXT-INDENT: 0pt; MARGIN-RIGHT: 3.6pt" align="left"><font style="DISPLAY: inline; FONT-SIZE: 14pt; FONT-FAMILY: Times New Roman">&#8220;We
consider that the capacity of NV-128 to trigger mTOR dephosphorylation leading
to caspase-independent cell death, in otherwise chemoresistant ovarian cancer
cells, opens new possibilities for the use of NV-128 as a potential addition to
conventional chemotherapy targeting ovarian cancer cells,&#8221; said Dr
Mor.</font></div>
      <div style="DISPLAY: block; MARGIN-LEFT: 0pt; TEXT-INDENT: 0pt; MARGIN-RIGHT: 0pt" align="justify">&#160;</div>
      <div style="DISPLAY: block; MARGIN-LEFT: 0pt; TEXT-INDENT: 0pt; MARGIN-RIGHT: 0pt" align="left"><font style="DISPLAY: inline; FONT-SIZE: 14pt; FONT-FAMILY: Times New Roman">In the
context of developing therapies indicated against late stage ovarian cancer, Dr
Mor said, &#8220;the demonstration of a functional caspase-independent cell death
pathway in apoptotic-resistant ovarian cancer cells is a key step to the
development of alternative targeted therapy for refractory
patients.&#8221;</font></div>
      <div style="DISPLAY: block; TEXT-INDENT: 0pt"><br></div>
      <div style="DISPLAY: block; MARGIN-LEFT: 0pt; TEXT-INDENT: 0pt; MARGIN-RIGHT: 0pt" align="left"><font style="DISPLAY: inline; FONT-SIZE: 14pt; FONT-FAMILY: Times New Roman">Structurally,
NV-128 is an analogue of triphendiol (NV-196) and phenoxodiol, both of which are
investigational drugs that have been licensed by Novogen to Marshall Edwards,
Inc.&#160;&#160;Phenoxodiol is currently in a multinational, multi-centre Phase
III clinical trial for patients with late stage ovarian
cancer.&#160;&#160;Triphendiol has recently been granted orphan drug status by
the FDA for pancreatic and bile duct cancers, and late stage
melanoma.</font></div>
      <div style="DISPLAY: block; MARGIN-LEFT: 0pt; TEXT-INDENT: 0pt; MARGIN-RIGHT: 0pt" align="justify">&#160;</div>
      <div style="DISPLAY: block; MARGIN-LEFT: 0pt; TEXT-INDENT: 0pt; MARGIN-RIGHT: 0pt" align="left"><font style="DISPLAY: inline; FONT-SIZE: 14pt; FONT-FAMILY: Times New Roman">&#8220;The
ability of our suite of analogues to invoke discreet modes of cell death
suggests that they have potential to be used synergistically, thereby inhibiting
alternative modes of cancer cell survival which may be invoked post
therapy.&#160;&#160;This could lower the incidence of residual disease
clinically,&#8221; said Professor Alan Husband, Group Director of Research, Novogen,
Limited.</font></div>
      <div style="DISPLAY: block; MARGIN-LEFT: 0pt; TEXT-INDENT: 0pt; MARGIN-RIGHT: 0pt" align="justify">&#160;</div>
      <div style="DISPLAY: block; MARGIN-LEFT: 0pt; TEXT-INDENT: 0pt; MARGIN-RIGHT: 10.8pt" align="left"><font style="DISPLAY: inline; FONT-SIZE: 14pt; FONT-FAMILY: Times New Roman">Unlike
analogues of rapamycin, like temsirolimus and everolimus, which target only
mTORC1, NV-128&#8217;s capacity to dephosphorylate mTOR enables it to inhibit both
mTORC1 and mTORC2 activity.&#160;&#160;This blocks growth factor driven
activation of AKT and the potential for development of
chemoresistance.&#160;&#160;Further, unlike NV-128, rapalogs invoke
caspase-mediated apoptosis making them less effective in those cancer cells that
have developed rapalog-resistance and have a dysfunctional apoptotic
cascade.</font></div>
      <div style="DISPLAY: block; MARGIN-LEFT: 0pt; TEXT-INDENT: 0pt; MARGIN-RIGHT: 0pt" align="justify">&#160;</div>
      <div style="DISPLAY: block; MARGIN-LEFT: 0pt; TEXT-INDENT: 0pt; MARGIN-RIGHT: 7.2pt" align="left"><font style="DISPLAY: inline; FONT-SIZE: 14pt; FONT-FAMILY: Times New Roman">Also
presented in Dr Alvero&#8217;s paper was a proof of concept study in an animal model
of drug resistant ovarian cancer, where it has been demonstrated that NV-128 not
only significantly retards tumour proliferation, but is more efficacious than
other standard of care drugs.&#160;&#160;It was also reported that
phosphorylated p70s6K was decreased, and endonucelase G was increased in tumours
taken from mice dosed with NV-128 thereby confirming that the NV-128 mechanism
of action <font style="DISPLAY: inline; FONT-STYLE: italic">in vivo </font>is
the same as that observed <font style="DISPLAY: inline; FONT-STYLE: italic">in
vitro, </font>and that both proteins can be employed as markers of NV-128
efficacy.</font></div>
      <div style="DISPLAY: block; MARGIN-LEFT: 0pt; TEXT-INDENT: 0pt; MARGIN-RIGHT: 0pt" align="justify">&#160;</div>
      <div style="DISPLAY: block; MARGIN-LEFT: 0pt; TEXT-INDENT: 0pt; MARGIN-RIGHT: 0pt" align="left"><font style="DISPLAY: inline; FONT-SIZE: 14pt; FONT-FAMILY: Times New Roman">&#8220;We are
just now beginning to realise the depth of oncology drug candidates that our
technology will uncover,&#8221; said Professor Husband. &#8220;We anticipate that refinement
of our proprietary molecular scaffold driven by computer-based molecular
modelling, will continue to yield novel derivatives not only indicated as
oncology leads, but also for cardiovascular and inflammatory
diseases.&#8220;</font></div>
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      <div style="DISPLAY: block; MARGIN-LEFT: 0pt; TEXT-INDENT: 0pt; MARGIN-RIGHT: 0pt" align="left"><font style="DISPLAY: inline; FONT-WEIGHT: bold; FONT-SIZE: 14pt; FONT-FAMILY: Times New Roman">About
NV<font style="DISPLAY: inline; FONT-FAMILY: Times New Roman">&#8208;</font>128:</font></div>
      <div style="DISPLAY: block; TEXT-INDENT: 0pt"><br></div>
      <div style="DISPLAY: block; MARGIN-LEFT: 0pt; TEXT-INDENT: 0pt; MARGIN-RIGHT: 0pt" align="left"><font style="DISPLAY: inline; FONT-SIZE: 14pt; FONT-FAMILY: Times New Roman">In
contrast to phenoxodiol and triphendiol, NV-128 has been shown to induce
caspase-independent DNA degradation and cancer cell death.&#160;&#160;It appears
that in conjunction with autophagy induction, NV-128 induces caspase
&#173;independent cell death via the AKT-mTOR pathway resulting in beclin
sequestration of Bcl-2, Bax up-regulation and mitochondrial
depolarization.&#160;&#160;As a consequence, endonuclease G translocates to the
nucleus where it initiates DNA degradation and cell death.&#160;&#160;This
offers an opportunity for use as a monotherapy in chemoresistant cancers and
enhanced efficacy against cancer targets less susceptible to
phenoxodiol.&#160;&#160;The option for co-administration of combinations of
these drugs is also under investigation to extend the potential therapeutic
range of this unique class of oncology compounds.</font></div>
      <div style="DISPLAY: block; MARGIN-LEFT: 0pt; TEXT-INDENT: 0pt; MARGIN-RIGHT: 0pt" align="justify">&#160;</div>
      <div style="DISPLAY: block; MARGIN-LEFT: 0pt; TEXT-INDENT: 0pt; MARGIN-RIGHT: 0pt" align="left"><font style="DISPLAY: inline; FONT-WEIGHT: bold; FONT-SIZE: 14pt; FONT-FAMILY: Times New Roman">About
Novogen Limited:</font></div>
      <div style="DISPLAY: block; TEXT-INDENT: 0pt"><br></div>
      <div style="DISPLAY: block; MARGIN-LEFT: 0pt; TEXT-INDENT: 0pt; MARGIN-RIGHT: 3.6pt" align="left"><font style="DISPLAY: inline; FONT-SIZE: 14pt; FONT-FAMILY: Times New Roman">Novogen
Limited (ASX: NRT; NASDAQ: NVGN) is an Australian biotechnology company that has
patented isoflavone technology for the treatment and prevention of degenerative
diseases and disorders. Over the past ten years, Novogen has conducted the
largest and most comprehensive isoflavone clinical testing programs in the
world.&#160;&#160;Novogen is involved in drug discovery and product development
for a range of degenerative disorders including cancer, cardiovascular diseases
and inflammatory diseases.&#160;&#160;The Company coordinates an international
clinical research and development program with external collaborators, hospitals
and universities. For more information, visit <font style="DISPLAY: inline; TEXT-DECORATION: underline">www.novogen.com</font>.</font></div>
      <div style="DISPLAY: block; MARGIN-LEFT: 0pt; TEXT-INDENT: 0pt; MARGIN-RIGHT: 0pt" align="justify">&#160;</div>
      <div style="DISPLAY: block; MARGIN-LEFT: 0pt; TEXT-INDENT: 0pt; MARGIN-RIGHT: 0pt" align="left"><font style="DISPLAY: inline; FONT-SIZE: 12pt; FONT-STYLE: italic; FONT-FAMILY: Times New Roman">Under
U.S. law, a new drug cannot be marketed until it has been investigated in
clinical trials and approved by the FDA as being safe and effective for the
intended use. Statements included in this press release that are not historical
in nature are "forward-looking statements" within the meaning of the "safe
harbor" provisions of the Private Securities Litigation Reform Act of 1995. You
should be aware that our actual results could differ materially from those
contained in the forward-looking statements, which are based on management's
current expectations and are subject to a number of risks and uncertainties,
including, but not limited to, our failure to successfully commercialize our
product candidates; costs and delays in the development and/or FDA approval, or
the failure to obtain such approval, of our product candidates; uncertainties in
clinical trial results; our inability to maintain or enter into, and the risks
resulting from our dependence upon, collaboration or contractual arrangements
necessary for the development, manufacture, commercialization, marketing, sales
and distribution of any products; competitive factors; our inability to protect
our patents or proprietary rights and obtain necessary rights to third party
patents and intellectual property to operate our business; our inability to
operate our business without infringing the patents and proprietary rights of
others; general economic conditions; the failure of any products to gain market
acceptance; our inability to obtain any additional required financing;
technological changes; government regulation; changes in industry practice; and
one-time events. We do not intend to update any of these factors or to publicly
announce the results of any revisions to these forward-looking
statements.</font></div>
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